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  • Faculty of Pharmacy
  • Course Structure Diagram with Credits
  • Pathology
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  • Will explain the science of pathology.
  • Defines pathology.
  • Explains the subfields of pathology and its relationship with other scientific disciplines.
  • Explains the concepts of homeostasis and cellular adaptation.
  • Will explain cellular adaptations.
  • Explains the concepts of homeostasis and cellular adaptation.
  • Explains the condition of hyperplasia.
  • Explains the condition of hypertrophy.
  • Explains the condition of atrophy.
  • Explains the condition of metaplasia.
  • Will explain cell injury.
  • Lists the causes of cell injury.
  • Lists the general principles of cell injury.
  • Lists the intracellular events affected by cell injury.
  • Lists the general biochemical mechanisms of cell injury.
  • Explains hypoxic, anoxic, and ischemic cell injury.
  • Explains ischemia/reperfusion injury.
  • Lists the molecules that increase and decrease in ischemic injury.
  • Explains cell injury caused by free radicals.
  • Lists the causes of free radical formation.
  • Lists the ways in which free radicals damage cells.
  • Lists the enzymes, vitamins, and minerals that protect cells against radicals.
  • Will explain intracellular accumulations.
  • Lists and explains intracellular accumulations (substances) that may cause cell injury.
  • Explains the causes of triglyceride accumulation in cells.
  • Lists pathological conditions observed as a result of triglyceride accumulation.
  • Explains the causes of cholesterol and cholesterol ester accumulation in cells.
  • Explains the causes of cholesterol and cholesterol ester accumulation in cells.
  • Explains the causes of protein accumulation in cells.
  • Lists pathological conditions observed as a result of protein accumulation.
  • Explains the causes of glycogen accumulation in cells.
  • Lists pathological conditions observed as a result of glycogen accumulation.
  • Defines pigment.
  • Lists exogenous pigments.
  • Lists endogenous pigments.
  • Provides examples of pathological conditions caused by pigments.
  • Defines pathological calcification.
  • Explains dystrophic calcification.
  • Explains metastatic calcification.
  • Will explain cellular aging and death.
  • Lists the metabolic changes observed in cellular aging.
  • Lists the morphological changes observed in cellular aging.
  • Explains necrosis.
  • Lists and explains the patterns of necrosis.
  • Lists the role of apoptosis in pathological events.
  • Will explain acute inflammation.
  • Defines inflammation.
  • Lists the differences between acute and chronic inflammation.
  • Lists the signs and causes of acute inflammation.
  • Lists the vascular changes that occur during acute inflammation.
  • Explains pathological conditions related to changes in vascular flow and diameter.
  • Lists and explains the cellular changes that occur during acute inflammation.
  • Explains pathological conditions related to margination and rolling.
  • Explains pathological conditions related to adhesion and transmigration.
  • Explains pathological conditions related to chemotaxis and activation.
  • Gives examples of chemotactic agents.
  • Lists the steps of phagocytosis formation.
  • Explains the mechanisms of degranulation, killing, and digestion.
  • Lists genetic or acquired leukocyte dysfunctions.
  • Lists the chemical mediators of inflammation and their sources.
  • Will explain chronic inflammation.
  • Lists the characteristics of chronic inflammation.
  • Lists the causes of chronic inflammation.
  • Lists the important cells and pathological conditions in chronic inflammation.
  • Will explain wound healing.
  • Explains the wound healing process.
  • Compares primary and secondary wound healing.
  • Lists the external and internal factors that affect wound healing.
  • Will explain edema.
  • Explains edema.
  • Explains the pathophysiology of edema.
  • Classifies the pathophysiological conditions caused by edema.
  • Will explain hyperemia and congestion.
  • Explains the concepts of hyperemia and congestion.
  • Explains the concept of hemorrhage.
  • Lists the pathophysiological conditions related to hemorrhage.
  • Will explain hemostasis and thrombosis.
  • Explains the concept of hemostasis.
  • Explains the concept of thrombosis.
  • Lists the elements of hemostasis and thrombosis.
  • Explains primary hemostasis.
  • Explains secondary hemostasis.
  • Explains the role of endothelin in hemostasis.
  • Explains the role of platelets in hemostasis.
  • Explains the coagulation cascade.
  • Explains the fibrinolytic cascade.
  • Explains Virchow's triad of thrombosis.
  • Lists the causes of endothelial damage.
  • Lists the causes of blood flow slowing.
  • Lists the causes of hypercoagulability.
  • Lists the conditions that thromboses can cause.
  • Explains venous thrombosis.
  • Explains arterial and cardiac thrombosis.
  • Will explain embolism.
  • Explains embolism.
  • Explains pulmonary thromboembolism.
  • Explains systemic thromboembolism.
  • Explains fat embolism.
  • Explains air embolism.
  • Explains amniotic fluid embolism.
  • Will explain shock.
  • Explains the condition of shock.
  • Lists the causes of shock.
  • Will explain neoplasia.
  • Explains neoplasia.
  • Defines benign tumor.
  • Defines malignant tumor.
  • Defines tumor components.
  • Names benign tumors based on parenchymal cell origin.
  • Names epithelial-derived benign tumors.
  • Names malignant tumors of mesenchymal tissues.
  • Defines mixed tumors and gives examples.
  • Defines mixed tumors and gives examples.
  • Defines teratoma tumors and gives examples.
  • Will explain the characteristics of benign and malignant tumors.
  • Lists the criteria by which benign and malignant tumors can be differentiated.
  • Explains the concepts of differentiation and anaplasia.
  • Defines anaplastic neoplasms and lists their characteristics.
  • Compares the growth rate of malignant and benign tumor cells.
  • Compares local invasion in malignant and benign tumor cells.
  • Explains the concept of metastasis.
  • Lists the pathways of metastasis in malignant neoplasms.
  • Will explain the epidemiology and etiology of cancer.
  • Explains cancer epidemiology.
  • Lists and provides examples of factors that cause cancer.
  • Lists the changes in malignant phenotypes.
  • Lists the effects of tumors on the host.
  • Explains cancer cachexia.
  • Explains paraneoplastic syndrome and provides examples.
  • Will explain environmental pollution.
  • Lists the causes of air pollution.
  • Lists indoor and outdoor air pollutants.
  • Lists the pathological conditions caused by air pollutants.
  • Lists industrial pollutants.
  • Lists the pathological conditions caused by industrial pollutants on organs/systems.
  • Defines pneumoconiosis.
  • Lists types of pneumoconiosis.
  • Lists the causes and sources of dust in pneumoconiosis.
  • Classifies and explains coal worker's pneumoconiosis.
  • Explains silicosis.
  • Explains asbestosis.
  • Explains berylliosis.
  • Will explain the harms of smoking.
  • Lists the substances found in tobacco smoke and their pathological effects.
  • Lists the tissues and organs affected by smoking.
  • Will explain damage caused by physical and chemical agents.
  • Provides examples of pathological effects caused by non-therapeutic chemical agents.
  • Provides examples of damage caused by physical agents.
  • Will explain protein-energy malnutrition.
  • Explains the appropriate diet components.
  • Compares primary and secondary malnutrition.
  • Explains protein-energy malnutrition.
  • Explains marasmus syndrome.
  • Explains kwashiorkor syndrome.
  • Will explain Anorexia Nervosa and Bulimia.
  • Explains the Anorexia Nervosa syndrome.
  • Explains the Bulimia syndrome.
  • Will explain vitamin and mineral deficiencies.
  • Lists vitamins and explains their deficiency syndromes.
  • Lists trace elements and explains their deficiency syndromes.
  • Will explain obesity.
  • Explains the pathological conditions that develop alongside obesity.

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